南臺灣醫學雜誌 (Record no. 7396)

MARC details
000 -LEADER
fixed length control field 00923cas a2200241 i 4500
001 - CONTROL NUMBER
control field .b47689109
003 - CONTROL NUMBER IDENTIFIER
control field YEEZEN
005 - DATE AND TIME OF LATEST TRANSACTION
control field 20230208135905.0
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION
fixed length control field 130904c20069999ch fr p r 0 0e0chi d
022 ## - INTERNATIONAL STANDARD SERIAL NUMBER
International Standard Serial Number 1991-4784 (平裝)
040 ## - CATALOGING SOURCE
Original cataloging agency NTU
Language of cataloging chi
Transcribing agency NTU
042 ## - AUTHENTICATION CODE
Authentication code nbic
066 ## - CHARACTER SETS PRESENT
Primary G0 character set $1
245 00 - TITLE STATEMENT
Title 南臺灣醫學雜誌
Remainder of title Medical journal of south Taiwan.
246 11 - VARYING FORM OF TITLE
Title proper/short title Medical journal of south Taiwan.
260 ## - PUBLICATION, DISTRIBUTION, ETC.
Place of publication, distribution, etc. 高雄市 :
Name of publisher, distributor, etc. 聯合出版醫院,
Date of publication, distribution, etc. 2006-
300 ## - PHYSICAL DESCRIPTION
Extent 冊 :
Other physical details 圖 ;
Dimensions 30公分
310 ## - CURRENT PUBLICATION FREQUENCY
Current publication frequency 半年刊
362 0# - DATES OF PUBLICATION AND/OR SEQUENTIAL DESIGNATION
Dates of publication and/or sequential designation 第1卷第1期(2006年6月)-
500 ## - GENERAL NOTE
General note 題名取自封面
500 ## - GENERAL NOTE
General note 本刊由高雄市國軍高雄總醫院, 高雄市立民生醫院, 臺南市郭綜合醫院, 屏東市寶建醫院, 屏東市行政院衛生署屏東醫院, 屏東縣東港安泰醫院, 高雄市阮綜合醫院, 高雄市國軍左營總醫院聯合出版
650 #7 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name entry element 醫學.
Source of heading or term lcstt.
650 #7 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name entry element 期刊.
Source of heading or term lcstt.
710 2# - ADDED ENTRY--CORPORATE NAME
Corporate name or jurisdiction name as entry element 聯合出版醫院
942 ## - ADDED ENTRY ELEMENTS (KOHA)
Source of classification or shelving scheme
Koha item type 期刊
Holdings
撤銷狀態 遺失狀態 分類號來源 損壞狀態 不外借 永久館藏地 現在館藏地 書架位置 採購日期 全部借出 完整索書號 條碼號 最後一次日期記錄 複本號 一般價格有效日期 Koha館藏類型 公開資訊
未註銷館藏 未遺失   未損毀 可外借 怡仁綜合醫院圖書館 怡仁綜合醫院圖書館   2023-02-08   Vol.16 N.1 F00047 2023-02-08 Vol.16 N.1 2023-02-08 期刊 Objective: This study investigated depression, self-care and quality of life in women with gynecological cancer in southern Taiwan. Methods: A cross-sectional design investigation method was adopted to facilitate the sampling of 350 gynecological cancer women were recruited through convenience sampling, at a teaching district hospital in southern Taiwan. Data were collected by delivering an interview survey of structured questionnaires. Outcome measurements included basic data, self-care scales, depression scales, and quality of life scales. Data were analyzed using SPSS 21.0 statistical software. Results: The results revealed a statistically significant negative correlation between self-care and occupation as well as between age and family type. Depression in women with cancer was negatively correlated with quality of life and positively correlated with self-care. These four factors accounted for 27.1% of the total variance in self-care; support from friends and poor sleep quality were two predictors of quality of life, accounting for 53.1% of the total variance. Conclusion: The results provide a reference for Clinical practice, administrative, and medical education can improve to the quality of life of women with gynecological cancer.
未註銷館藏 未遺失   未損毀 可外借 怡仁綜合醫院圖書館 怡仁綜合醫院圖書館   2023-02-08   Vol.16 N.2 F00048 2023-02-08 Vol.16 N.2 2023-02-08 期刊 Objective: Hepatocarcinogenesis is a complex process and hard to understand. By an ideal mathematical calculation in 2009, I found the hepatocarcinogenesis might be constituted by 6 molecular pathway permutations. By understanding the progress of hepatocellular carcinoma drugs in the past 10 years, I will verify its correctness and will explain the current status of hepatocellular carcinoma drug treatment. Methods: Review associated 2010-2020 Pub-med articles about advanced hepatocellular carcinoma drugs treatment and the IMbrave 150 study. Results: The IMbrave 150 study confirms my previous theoretical prediction "if we use 1 or 2 key molecular targeted therapies, the disease control rate will be 41-51% and 59-76% respectively." Both anti-PD1/PDL1 and anti-VEGF can treat 2 targeted pathways of the 6 molecular pathways of hepatocarcinogenesis. Conclusion: The ideal mathematical calculation/model in 2009 by Dr. Huang et al. could explain the progress of medical treatment of hepatocellular carcinoma in the past ten years and it could predict future combinations of therapeutic development and direction.
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